一个额外的基因增加了唐氏综合症小鼠大脑中的抑制信号

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• 1. Apigenin as a Candidate Prenatal Treatment for Trisomy 21: Effects in Human Amniocytes and the Ts1Cje Mouse Model.

  
  

  Faycal Guedj,Ashley E Siegel,Jeroen L A Pennings,Fatimah Alsebaa,Lauren J Massingham,Umadevi Tantravahi,Diana W Bianchi

  Prenatal Genomics and Therapy Section, Medical Genomics and Metabolic Genetics Branch, National Human Genome Research Institute, National Institutes of Health, Bethesda, MD 20892, USA; Mother Infant Research Institute, Tufts Medical Center and Tufts Children's Hospital, Boston, MA 02111, USA. Electronic address: faycal.guedj@nih.gov.

  faycal.guedj@nih.gov

  Am J Hum Genet (P 1537-6605 E 0002-9297) H指数：280 2020 年 107 卷 5 期 911-931 页

  PMID：33098770 相似文献

  文摘 DOI链接 Elsevier Science

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  2. Overexpression of DYRK1A inhibits choline acetyltransferase induction by oleic acid in cellular models of Down syndrome.

  
  

  Maruan Hijazi,Cristina Fillat,José M Medina,Ana Velasco

  Instituto de Neurociencias de Castilla y León (INCYL), Universidad de Salamanca, (IBSAL), Spain.

  
  

  Exp Neurol (P 1090-2430 E 0014-4886) H指数：168 2013 年 239 卷 229-34 页

  PMID：23124096 相似文献

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